Monosomal karyotype as an adverse prognostic factor in patients with acute myeloid leukemia treated with allogeneic hematopoietic stem-cell transplantation in first complete remission: a retrospective survey on behalf of the ALWP of
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Overexpression of the human DEK oncogene reprograms cellular metabolism and promotes glycolysis
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The role of HLA variation in lymphoma aetiology and survival - Zhong - 2019 - Journal of Internal Medicine - Wiley Online Library
Epithelial‑mesenchymal transition in colorectal carcinoma cells is mediated by DEK/IMP3
Epithelial‑mesenchymal transition in colorectal carcinoma cells is mediated by DEK/IMP3
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Epithelial‑mesenchymal transition in colorectal carcinoma cells is mediated by DEK/IMP3
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Overexpression of the human DEK oncogene reprograms cellular metabolism and promotes glycolysis
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DEK-CAN molecular monitoring of myeloid malignancies could aid therapeutic stratification | Leukemia
Efficacy of catheter-based renal denervation in the absence of antihypertensive medications (SPYRAL HTN-OFF MED Pivotal): a multicentre, randomised, sham-controlled trial - The Lancet
DEK-CAN molecular monitoring of myeloid malignancies could aid therapeutic stratification | Leukemia
Epithelial‑mesenchymal transition in colorectal carcinoma cells is mediated by DEK/IMP3
![Genomic signatures of globally enhanced gene duplicate accumulation in the megadiverse higher Diptera fueling intralocus sexual conflict resolution [PeerJ]](https://dfzljdn9uc3pi.cloudfront.net/2020/10012/1/fig-2-full.png "Genomic signatures of globally enhanced gene duplicate accumulation in the megadiverse higher Diptera fueling intralocus sexual conflict resolution [PeerJ]")
Genomic signatures of globally enhanced gene duplicate accumulation in the megadiverse higher Diptera fueling intralocus sexual conflict resolution [PeerJ]
Significance of DEK overexpression for the prognostic evaluation of non-small cell lung carcinoma
Immiscible inclusion bodies formed by polyglutamine and poly(glycine-alanine) are enriched with distinct proteomes but converge in proteins that are risk factors for disease and involved in protein degradation
